Cell-Mediated Immunity in Glomerular Disease
1991
Our understanding of the role of the
immune systemin modulating glomerular injury continues to evolve. The participation of antibody, complement, and neutrophils alone cannot completely explain many aspects of the pathways of experimental and human
glomerulonephritis. The elements of the
immune systemthat mediate cellular immunity, T lymphocytes and monocytes, also appear to be important effectors of
glomerulonephritis. An understanding of the role of cell-mediated immunity in renal disease is essential in designing therapies to interrupt immunologically sustained glomerular damage.
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